H. pylori genome/methylome project in Japan
We have decoded genomes/methylomes of over 150 H. pylori strains by Single-Molecule Real-Time (SMRT) sequencing in Pacbio machines in collaboration with National Institute of Genetics, National Institute for Basic Biology, and Pacificbioscience of California.
Helicobacter pylori infection induces abnormal expression of proangiogenic genes and microRNAs in gastric cell lines
H. pylori infection can induce angiogenesis in the pre-neoplastic cascade of events that culminates in gastric cancer, which may have important functional implications for the etiology of this disease.
Isolation frequency of H. pylori and non-H. pylori bacteria from gastric biopsies of patients with chronic gastritis, gastric ulcer and gastric cancer
Recent studies show that, in addition to Helicobacter pylori, other bacterial species such as Proteus mirabilis, Citrobacter freundii, Klebsiella pneumoniae, Enterobacter cloacae and Staphylococcus aureus are found in the gastric mucosa, either in co-infection with H. pylori or not.
Rapid evolution of AlpA adhesin of Helicobacter pylori in Colombia
In order to survive, H. pylori must adheres the gastric epithelial cells of its human host. For this purpose, the bacterium employs an array of adhesins, for example AlpA.
Gastric Carcinoma profile characterization: retrospective analysis of 275 cases submitted to gastrectomy at Clinics Hospital of UFMG in the last 12 years
Gastric carcinoma (GC) is the third leading cause of cancer death among malignant tumors worldwide. It is considered a multifactorial disease, presenting high mortality rates in both sexes.
H. pylori, EBV, and CMV in Mexican patients with chronic gastritis and gastric cancer
Helicobacter pylori (H. pylori) infection is the main risk factor for gastric cancer, however, evidence is growing that infectious agents such as Epstein-Barr Virus (EBV) and Cytomegalovirus (CMV) are also related to this malignancy.