Ex vivo emission of volatile organic compounds from gastric cancer and non-cancerous tissue
Volatile organic compound (VOC) detection in the exhaled air has a potential for gastric cancer screening. Questions of the VOC origin and their biological relevance still require to be answered.
THE PREVALENCE OF DECREASED PEPSINOGEN LEVELS AND HELICOBACTER PYLORI ANTIBODIES IN KAZAKHSTAN
Serum pepsinogen (Pg) I and the ratio between PgI and PgII (PgI/PgII) are related to the histological and functional status of the gastric mucosa. Low serum PgI and PgI/PgII values are biomarkers for atrophic gastritis.
Molecular evolution by functional divergence of the p55 binding domain of VacA in Colombian isolates of Helicobacter pylori
Latin America was affected by several human migratory waves during the colonisation period. Humans arrived at the continent with a new subset of Helicobacter pylori, which replaced the native species.
H. pylori vacA and cagA genotypes and regulatory T cells in patients with chronic gastritis and gastric cancer
In the human gastric mucosa, Helicobacter pylori activates mechanisms that induce the preferential differentiation of regulatory T cells (Treg). Treg cells are CD4+, CD25+, and FOXP3+; have suppressive activity of inflammation and facilitate the immune escape of tumor cells.
A LONGITUDINAL STUDY OF CELL-FREE DNA OF REPRIMO METHYLATED, PEPSINOGENS AND HELICOBACTER PYLORI IN PLASMA AS A NON-INVASIVE RISK-ASSESSMENT FOR OLGA STAGING
Gastric cancer (GC) is the fifth most common cause of cancer death worldwide with approximately one million cases diagnosed annually.
Helicobacter pylori vacA s1m1/cagA+ promotes increased expression of IFN-γ and IL-17A in human mononuclear cells
The persistent infection of the gastric mucosa by Helicobacter pylori (H. pylori) and the resulting chronic inflammation are a risk factor for gastric carcinogenesis.